Descriptor English: | Porphyria Cutanea Tarda | ||||
Descriptor Spanish: |
Porfiria Cutánea Tardía
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Descriptor Portuguese: | Porfiria Cutânea Tardia | ||||
Descriptor French: | Porphyrie cutanée tardive | ||||
Tree number(s): |
C06.552.830.100 C16.320.850.742.250 C17.800.827.742.250 C18.452.811.400.250 |
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RDF Unique Identifier: | https://id.nlm.nih.gov/mesh/D017119 | ||||
Scope note: | An autosomal dominant or acquired porphyria due to a deficiency of UROPORPHYRINOGEN DECARBOXYLASE in the LIVER. It is characterized by photosensitivity and cutaneous lesions with little or no neurologic symptoms. Type I is the acquired form and is strongly associated with liver diseases and hepatic toxicities caused by alcohol or estrogenic steroids. Type II is the familial form. |
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Allowable Qualifiers: |
BL blood CF cerebrospinal fluid CI chemically induced CL classification CN congenital CO complications DG diagnostic imaging DH diet therapy DI diagnosis DT drug therapy EC economics EH ethnology EM embryology EN enzymology EP epidemiology ET etiology GE genetics HI history IM immunology ME metabolism MI microbiology MO mortality NU nursing PA pathology PC prevention & control PP physiopathology PS parasitology PX psychology RH rehabilitation RT radiotherapy SU surgery TH therapy UR urine VE veterinary VI virology |
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Previous Indexing: |
Porphyria (1966-1992) |
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Public MeSH Note: | 95; was PORPHYRIA, CUTANEA TARDA 1993-94 |
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Online Note: | use PORPHYRIA CUTANEA TARDA to search PORPHYRIA, CUTANEA TARDA 1993-94 |
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History Note: | 95; was PORPHYRIA, CUTANEA TARDA 1993-94 |
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DeCS ID: | 30608 | ||||
Unique ID: | D017119 | ||||
Documents indexed in the Virtual Health Library (VHL): | Click here to access the VHL documents | ||||
Date Established: | 1993/01/01 | ||||
Date of Entry: | 1992/05/22 | ||||
Revision Date: | 2018/06/30 |
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Porphyria Cutanea Tarda
- Preferred
Concept UI |
M0026011 |
Scope note | An autosomal dominant or acquired porphyria due to a deficiency of UROPORPHYRINOGEN DECARBOXYLASE in the LIVER. It is characterized by photosensitivity and cutaneous lesions with little or no neurologic symptoms. Type I is the acquired form and is strongly associated with liver diseases and hepatic toxicities caused by alcohol or estrogenic steroids. Type II is the familial form. |
Preferred term | Porphyria Cutanea Tarda |
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