Descriptor English: | Hypoaldosteronism | ||||||
Descriptor Spanish: |
Hipoaldosteronismo
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Descriptor Portuguese: | Hipoaldosteronismo | ||||||
Descriptor French: | Hypoaldostéronisme | ||||||
Entry term(s): |
Acidosis, Renal Tubular Type IV Hypoaldosteronism, Hyporeninemic Hyporeninemic Hypoaldosteronism Renal Tubular Acidosis, Type IV Type IV Renal Tubular Acidosis |
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Tree number(s): |
C19.053.500.480 |
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RDF Unique Identifier: | https://id.nlm.nih.gov/mesh/D006994 | ||||||
Scope note: | A congenital or acquired condition of insufficient production of ALDOSTERONE by the ADRENAL CORTEX leading to diminished aldosterone-mediated synthesis of Na(+)-K(+)-EXCHANGING ATPASE in renal tubular cells. Clinical symptoms include HYPERKALEMIA, sodium-wasting, HYPOTENSION, and sometimes metabolic ACIDOSIS. |
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Annotation: | do not confuse with HYPERALDOSTERONISM or PSEUDOHYPOALDOSTERONISM |
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Allowable Qualifiers: |
BL blood CF cerebrospinal fluid CI chemically induced CL classification CN congenital CO complications DG diagnostic imaging DH diet therapy DI diagnosis DT drug therapy EC economics EH ethnology EM embryology EN enzymology EP epidemiology ET etiology GE genetics HI history IM immunology ME metabolism MI microbiology MO mortality NU nursing PA pathology PC prevention & control PP physiopathology PS parasitology PX psychology RH rehabilitation RT radiotherapy SU surgery TH therapy UR urine VE veterinary VI virology |
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Previous Indexing: |
Adrenal Gland Hypofunction (1966-1987) Aldosterone (1966-1987) |
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Public MeSH Note: | 88 |
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History Note: | 88 |
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DeCS ID: | 22918 | ||||||
Unique ID: | D006994 | ||||||
Documents indexed in the Virtual Health Library (VHL): | Click here to access the VHL documents | ||||||
Date Established: | 1988/01/01 | ||||||
Date of Entry: | 1987/03/13 | ||||||
Revision Date: | 2006/07/05 |
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Hypoaldosteronism
- Preferred
Hypoaldosteronism, Hyporeninemic
- Narrower
Concept UI |
M0010887 |
Scope note | A congenital or acquired condition of insufficient production of ALDOSTERONE by the ADRENAL CORTEX leading to diminished aldosterone-mediated synthesis of Na(+)-K(+)-EXCHANGING ATPASE in renal tubular cells. Clinical symptoms include HYPERKALEMIA, sodium-wasting, HYPOTENSION, and sometimes metabolic ACIDOSIS. |
Preferred term | Hypoaldosteronism |
Concept UI |
M0010888 |
Scope note | A selective aldosterone deficiency resulting from diminished RENIN-generated ANGIOTENSIN II, a key stimulus to aldosterone secretion. Previously, it was called type IV renal tubular acidosis. |
Preferred term | Hypoaldosteronism, Hyporeninemic |
Entry term(s) |
Acidosis, Renal Tubular Type IV Hyporeninemic Hypoaldosteronism Renal Tubular Acidosis, Type IV Type IV Renal Tubular Acidosis |
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